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Long-lived C. elegans daf-2 mutants are resistant to bacterial pathogens

Science. 2003 Jun 20;300(5627):1921. doi: 10.1126/science.1080147.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Alleles
  • Animals
  • Caenorhabditis elegans / genetics
  • Caenorhabditis elegans / immunology
  • Caenorhabditis elegans / microbiology*
  • Caenorhabditis elegans / physiology*
  • Caenorhabditis elegans Proteins / genetics
  • Caenorhabditis elegans Proteins / physiology
  • Enterococcus faecalis / pathogenicity*
  • Enterococcus faecalis / physiology
  • Escherichia coli / pathogenicity*
  • Escherichia coli / physiology
  • Forkhead Transcription Factors
  • Longevity
  • Mutation
  • Phenotype
  • Phosphatidylinositol 3-Kinases / genetics
  • Phosphatidylinositol 3-Kinases / physiology
  • Receptor, Insulin / genetics
  • Receptor, Insulin / physiology*
  • Staphylococcus aureus / pathogenicity*
  • Staphylococcus aureus / physiology
  • Transcription Factors / genetics
  • Transcription Factors / physiology

Substances

  • Caenorhabditis elegans Proteins
  • Forkhead Transcription Factors
  • Transcription Factors
  • daf-16 protein, C elegans
  • AGE-1 protein, C elegans
  • DAF-2 protein, C elegans
  • Receptor, Insulin