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Three types of pathological cold antibodies can all be ''[[acquired]]'' <ref name=" 1956 pp. 221–40">{{cite journal | last=WIENER | first= AS | last2=UNGER | first2= LJ | last3=COHEN | first3= L | last4=FELDMAN | first4= J | title=Type-specific cold auto-antibodies as a cause of acquired hemolytic anemia and hemolytic transfusion reactions: biologic test with bovine red cells. | journal=Annals of internal medicine | volume=44 | issue=2 | year=1956 | issn=0003-4819 | pmid=13292836 | pages=221–40}}</ref><ref name="Mehrotra 1960 pp. 265–271">{{cite journal | last=Mehrotra | first=TN | title=Immunological Identification of the Pathological Cold Auto-Antibodies of Acquired Haemolytic Anaemia as β2M-Globulin | journal=Immunology | volume=3 | issue=3 | year=1960 | pmid= | pmc=1424011 | pages=265–271}}</ref><ref>[https://www.webmd.com/a-to-z-guides/autoimmune-hemolytic-anemia Acquired Autoimmune Hemolytic Anemia]</ref><ref name="Allgood Chaplin 1967 pp. 254–73">{{cite journal | last=Allgood | first=JW | last2=Chaplin | first2=H Jr | title=Idiopathic acquired autoimmune hemolytic anemia. A review of forty-seven cases treated from 1955 through 1965. | journal=The American journal of medicine | volume=43 | issue=2 | year=1967 | issn=0002-9343 | pmid=6034957 | pages=254–73}}</ref>.
===Complement===
[[Complement activation]] plays a definitive but limited role in warm-antibody AIHA (w-AIHA), whereas primary cold agglutinin disease (CAD), secondary cold agglutinin syndrome (CAS), and paroxysmal cold hemoglobinuria (PCH) are entirely complement-dependent disorders. <ref name="Berentsen Sundic 2015 pp. 1–11"/>
==Reference==
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